Due to the mortality and morbidity of cardiovascular disease, there is great interest in the flow dynamics and fluid mechanical forces in stenotic vessels. Critical stress conditions both on [1–2] and within [3] atherosclerotic plaque deposits have been implicated in plaque rupture. The presence of atherosclerotic lesions constricting and accelerating blood flow changes the wall behavior, and can also trigger intermittent, unstable, or transitional flows [4–5]. The current work evaluates the effect of incorporating flow turbulence and wall compliance in hemodynamic models.

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